The most common cardiac arrhythmia in clinical practise is atrial fibrillation (AF). Many research have been conducted to determine what causes AF to develop, but the issue has yet to be resolved. Based on the cessation of atrial mechanical systole, atrial fibrillation has lately been hypothesised as a protective physiological mechanism. In pathological conditions, this lowers the pressure in the pulmonary veins and alveolar capillaries, lowering the risk of pulmonary congestion and edoema. This hypothesis explains the link between left ventricle (LV) dysfunction and atrial fibrillation (AF), aids in the development of algorithms for the relationship between AF and LV dysfunction, as well as other diseases and conditions, suggests a new approach to locating the source of AF, and demonstrates the atriums' protective role in AF. the progression of LV dysfunction The idea is well-supported by known facts and phenomena relevant to AF. The proposed idea may provide insight into the progression of AF. In the pulmonary veins, each person has a defensive physiological system that is most likely genetically designed.
Author(S) Details
Vladimir Tilman
The Sheba Medical Center, Rehabilitation Hospital, Ramat Gan, Israel.
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