Sunday, 1 February 2026

Quantitative Study on Immunity and Tissue Regenerative Capacity in Hemodialysis Patients with Diabetes: General Characteristics and Paradoxical Aspects | Chapter 3 | An Overview of Disease and Health Research Vol. 9

 

Aims: This chapter aims to present the results of studies on intestinal microbiota, innate and acquired immune responses, and tissue regenerative capacity in hemodialysis patients with T2DM.

 

Study Design: It is a quantitative study that used two types of biological samples: blood and faeces.

 

Place and Duration of Study: Institute of Virology and University Research Institute laboratories, between January and June 2024. 

 

Methodology: The study was conducted on 60 patients divided into two groups: the test group (patients with ESRD and T2DM) and the control group (ESRD only). The intestinal microbiota was investigated using a genetic study. As markers for innate immunity (inflammation), the IL-6, sIL-6R, IL-1β, TNFα, IL-10, and NGAL serum levels (ELISA kits). As a cellular immunity marker, TNFβ/LTα was investigated. Regenerative capacity was studied using NT-3 (this is the first study to do so) and VEGFβ (another marker that is scarcely found in this patient category) (ELISA kits).

 

Results: The results proved a marked polarisation. IL-6 and sIL-6R presented significant increases in both groups, especially in diabetics. IL-6 generates trans-signalling through sIL-6R, with proinflammatory and anti-regenerative effects, confirmed through a significant reduction in NT-3 and VEGF-β. Statistical analysis has allowed us to conclude that the high level of IL-6 significantly influences IL-1β, TNF-β, NT-3, VEGF-β and IL-10 behaviour. But we were very surprised to find unexpected immunological changes and tissue regenerative capacity in an 82-year-old female patient, diagnosed with insulin-requiring T2DM, with multiple complications, including ESRD.

 

Conclusions: Although this research is in its early stages, it generates important conclusions and directions for further research. Regarding the particular case presented, we support the following causal hypotheses: genetically conditioned IL-6 overproduction (possibly acquired post-infection), together with its predominant anti-inflammatory and pro-regenerative signalling through IL-6R membrane receptors; the possibility that the extremely high levels of growth factors predict severe decompensation of the two associated diseases.

 

 

Author(s) Details

 

 

Mihaela Gheorghiu
Pathophysiology and Immunology Department, “Carol Davila” University of Medicine and Pharmacy, Bucharest, Romania.

 

Maria-Florina Trandafir
Pathophysiology and Immunology Department, “Carol Davila” University of Medicine and Pharmacy, Bucharest, Romania.

 

Coralia Bleotu
“Stefan S. Nicolau” Institute of Virology, Bucharest, Romania.

 

Octavian Savu
“N.C. Paulescu” National Institute of Diabetes, Nutrition and Metabolic Diseases, Bucharest, Romania and Doctoral School of “Carol Davila”, University of Medicine and Pharmacy, Bucharest, Romania.

 

Please see the book here :- https://doi.org/10.9734/bpi/aodhr/v9/7002

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