Physical Education and Physical Fitness Across Development: A Neurodevelopmental Perspective on Childhood and Adolescence |Chapter 2 | An Overview of Disease and Health Research Vol. 10

 

Physical education is a fundamental component of childhood and adolescent development, contributing not only to physical fitness but also to cognitive, emotional, and psychosocial maturation. Contemporary evidence from developmental neuroscience highlights adolescence as a sensitive period of heightened neuroplasticity, during which physical activity exerts profound effects on brain structure and function. School-based physical education provides a unique environment that combines movement, novelty, skill acquisition, and peer interaction; key drivers of neural adaptation during this developmental “grey zone.” This chapter examines physical education as a neurodevelopmentally active process, synthesising evidence on its role in enhancing physical fitness alongside executive function, emotional regulation, motivation, and mental well-being. By integrating perspectives from exercise science and adolescent neuroscience, the chapter emphasises the need to design physical education programs that align with brain maturation processes to optimise both physical and mental health outcomes in children and adolescents.

 

 

Author(s) Details

Akanksha Nagar

BFUHS, India and Department of Physiotherapy, Faculty of Health and Allied Sciences, Guru Kashi University, Bathinda, Punjab, 151302, India.

 

Himanshu Kaushal
Department of Neurology, Guru Gobind Singh Medical College & Hospital, Faridkot, Punjab, 151203, India.

 

Jaspreet Vij
Institute of Physiotherapy, University Regional Centre, Sri Goindwal Sahib, Baba Farid University of Health Sciences, Punjab, 151203, India.

 

Please see the book here :- https://doi.org/10.9734/bpi/aodhr/v10/7018

Determining the Effect of High Carbohydrate Diets on Brain Composition and Senescence in Aging Hyperinsulinemic Obese LA/Ntul//-cp Rats | Chapter 12 | Novel Research Aspects in Medicine and Medical Science Vol. 2

 This division determine the effect of corpulent phenotype and the carbohydrate type resulted in alterations in intellect composition in the corpulent phenotype of the congenic LA/Ntul//-cp rat, groups (n= 8 rats/group) of male littermate lean and corpulent rats were fed standardized isocaloric diets holding 54% (w/w) cornstarch (ST diet) or 54% (w/w) sucrose (SUC diet) from 1 just before 10.5 ± 0.5 months of age. Malnutrition is a extensive global public health issue that donates to several able to be consumed-related non-able to be contracted illnesses. In reduced-income nations, adult food is frequently missed. In low- and middle-proceeds nations, both two-fold malnutrition (over and undernutrition) and food transition survive. The current popular emphasis on liquid produced by mammals diets has resulted in an upwards shift in abstinence from food carbohydrate use. The strain's obese phenotype exhibits hypertrophic-hyperplastic obesity all along early postweaning development and early beginning chronic hyperinsulinemia outside NIDDM. The total fat, protein, and DNA of typical aliquots of dissected brain tissues were resolved. When fed the SUC meat as opposed to the ST diet, frame weights of obese >> lean mammals were higher in both genotypes. In rats augment the SUC diet compared to the ST diet, intellect mass was a little smaller in lean individuals distinguished to fat individuals. Brain total Protein and DNA content of lean rats were > corpulent rats and were modestly Lower in SUC than ST augment rats in both phenotypes, but the percent of lipid content was equivalent to brain bulk. The decreased intelligence mass was characterized by balanced decreases in total lipid, brain protein and intellect DNA content and were further impaired when augment the insulinogenic sucrose diet. Total body fat bulk of obese was considerably greater than happen in lean littermates and was only modestly better in SUC than ST fed rats in both phenotypes. These results signify that brain development and cellular growth is impaired in the aging, hyperinsulinemia-likely obese phenotype concerning this strain, were further impaired when augment SUC than ST diets, and the decreased brain limits were likely associated with happening of a chronic neuronal angering syndrome common to overdone fat accretion and corpulence, resulting in impulsive brain senescence.

Author(s) Details:

Orien L. Tulp,
Colleges of Medicine and Graduate Studies, University of Science Arts and Technology, Montserrat, MSR1110, British West Indies and The Einstein Medical Institute, NPB, Florida-34040, USA.

Please see the link here: https://stm.bookpi.org/NRAMMS-V2/article/view/11843