Introduction: Every epidemic that breaks out could cause confusion and fear in society. Little is known regarding behavioural changes and health condition responses, despite the dramatic warning in the media. There is an extremely strong correlation between the onset of cardiovascular disease and the pandemic.
Aim: The goal of the study was to find out how the pandemic's
stress affected the heart's regular physiological processes.
Methodology: Information about the potential effects of
psychological stress brought on by the pandemic on cardiac physiological
function was gathered from several databases. Using the various databases,
studies that examined the relationship between stress on CVDs or the emergence
of the pandemic and the cardiovascular system were found. A total of 37 studies
were chosen for full assessment out of the more than 200 abstracts that were
screened to ascertain whether all inclusion requirements were satisfied. Among
these studies, one focused on evaluating the stress levels of front-line
medical staff in China, particularly those working at a provincial hospital
during the coronavirus outbreak and severe acute respiratory syndrome (SARS).
Discussion: As a result, a study revealed that medical staff
members experienced significantly higher levels of moderate and severe anxiety
than their administrative counterparts. Prior research on severe acute
respiratory syndrome emphasised the importance of increasing awareness of
mitigating strategies. Because a larger perceived danger of infection was more
likely to result in an increase in cautionary events in contrast to
contamination, this strategy was taken into consideration with the underlying
insights of risk and anxiety. The implementation of control measures raises the
stress level, which may cause CVD to develop.
Conclusion: There is adequate evidence that illustrates the
physiological manifestation of the respiratory system and obstruction of the
blood flow that might lead to the development of CVD. If not properly
controlled might lead to death due to a heart attack.
Author(s)
Details
Matome
M Sekhotha
Department of Physiology and Environmental Health, Faculty of
Science and Agriculture, School of Molecular and Life Sciences, University of
Limpopo, Private Bag X 1106, Sovenga 0727, South Africa.
Tlou R
Senosha
Department of Physiology and Environmental Health, Faculty of
Science and Agriculture, School of Molecular and Life Sciences, University of
Limpopo, Private Bag X 1106, Sovenga 0727, South Africa.
Maropeng
R Ntsoane
Department of Physiology and Environmental Health, Faculty of
Science and Agriculture, School of Molecular and Life Sciences, University of
Limpopo, Private Bag X 1106, Sovenga 0727, South Africa.
Please
see the book here:- https://doi.org/10.9734/bpi/aodhr/v3/5511
