Showing posts with label cortical spreading depolarizations. Show all posts
Showing posts with label cortical spreading depolarizations. Show all posts

Friday, 8 July 2022

Improving Saudi EFL Reading Skills with an English Novel | Chapter 6 | Current Research in Language, Literature and Education Vol. 7

A three-week intensive reading intervention based on Charles Dickens' novel Great Expectations was the focus of this mixed-methods study, which examined how 10 Saudi EFL university students regarded the utilisation of real literary resources. The majority of sessions are focused on a teacher-centered approach, and students are typically assigned an inactive role in the learning process in Saudi university EFL reading classes, which frequently adhere to traditional teaching techniques. The participants were divided into two groups at random: an experimental group and a control group. The study used semi-structured interviews to acquire qualitative data in addition to a pre- and post-test to gauge reading improvement. According to the results, participants felt positively about using interactive training with real literary resources, such books, in the Saudi EFL reading classroom. Despite this optimistic outlook, participants experienced a number of challenges during their first participation in the trial, mostly because they lacked vocabulary and were used to reading such a lengthy material.


Author(s) Details:

Hammad Ali Alshammari,
English Department, College of Arts, Jouf University, Kingdom of Saudi Arabia.

Elsayed Abdallah Ahmed,
English Department, College of Arts, Jouf University, Kingdom of Saudi Arabia.

Please see the link here: https://stm.bookpi.org/CRLLE-V7/article/view/7539

Tuesday, 19 October 2021

Cortical Spreading Depolarizations in the Context of Subarachnoid Hemorrhage: A Review | Chapter 17 | Recent Developments in Medicine and Medical Research Vol. 3

 One of the most common consequences of spontaneous subarachnoid haemorrhage is delayed cerebral ischemia (DCI), which is caused by cortical spreading depolarizations (CSDs). Waves of neuronal and glial depolarizations in the cortex cause a disruption of neuronal ionic homeostasis, resulting in potassium efflux and sodium and calcium inflow. After aneurysmal subarachnoid haemorrhage, CSDs in the cerebral cortex are involved in the aetiology of both acute and delayed cerebral ischemia (aSAH). CSDs cause microvascular vasoconstriction and, as a result, hypoperfusion and the propagation of ischemia in injured brain areas and brain areas at risk, such as those next to subarachnoid haemorrhage (SAH). A number of research have been conducted in order to reduce secondary damage that develop hours to days following an acute insult. Due to its pharmacodynamic effects at the cellular level, ketamine, which was previously prohibited in the neurosurgical population due to the possibility of producing intracranial hypertension, has re-emerged as a possible neuroprotective medication. Anti-inflammatory processes, as well as controls over microthrombosis and cell apoptosis, as well as modification of brain excitotoxicity and CSDs, are among these effects. Retrospective studies corroborated the benefits of ketamine on CSD control and, as a result, DCI in patients with SAH, but no change in clinical outcome was observed. In prospective randomised studies, the effect of ketamine on the occurrence/development of DCI needs to be established.


Author (S) Details

Leandro Custódio do Amaral

Neurosurgeon, Rede Mater Dei de Saúde, Hospital Metropolitano Odilon Behrens and Fundação Benjamin Guimarães, Hospital da Baleia, Brazil.



View Book :- https://stm.bookpi.org/RDMMR-V3/article/view/4164